Presentation | 2012-06-23 Atypical Language Acquisition in Williams Syndrome and the Issue of Language Modularity Fusa KATADA, |
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Abstract(in Japanese) | (See Japanese page) |
Abstract(in English) | Williams syndrome (WS) is a multisystem genetic disorder caused by microdeletion of about 26 genes, most notably ELN, from chromosome #7 (7q11.23). Despite the syndrome being neurodevelopmental, WS adolescents and adults are marked by dissociations within and across domains of higher cognitive functioning, which raise important issues concerning language acquisition and cognitive modularity. This paper reviews two contrastive positions surrounding the issues: nativist and neuroconstructivist views. It then introduces a rare case with WS in a Japanese individual, which indicates unusual plasticity for language acquisition, and examines logical correlations between atypicality and modularity. |
Keyword(in Japanese) | (See Japanese page) |
Keyword(in English) | Williams syndrome / Atypical language acquisition / Plasticity for language acquisition / Cognitive dissociations / Cognitive modularity |
Paper # | TL2012-4 |
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Committee | TL |
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Conference Date | 2012/6/16(1days) |
Place (in Japanese) | (See Japanese page) |
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Topics (in Japanese) | (See Japanese page) |
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Paper Information | |
Registration To | Thought and Language (TL) |
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Language | JPN |
Title (in Japanese) | (See Japanese page) |
Sub Title (in Japanese) | (See Japanese page) |
Title (in English) | Atypical Language Acquisition in Williams Syndrome and the Issue of Language Modularity |
Sub Title (in English) | |
Keyword(1) | Williams syndrome |
Keyword(2) | Atypical language acquisition |
Keyword(3) | Plasticity for language acquisition |
Keyword(4) | Cognitive dissociations |
Keyword(5) | Cognitive modularity |
1st Author's Name | Fusa KATADA |
1st Author's Affiliation | Faculty of Science and Engineering, Waseda University() |
Date | 2012-06-23 |
Paper # | TL2012-4 |
Volume (vol) | vol.112 |
Number (no) | 103 |
Page | pp.pp.- |
#Pages | 5 |
Date of Issue |